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Semax is a corticotropin like peptide that can penetrate the blood-brain barrier and form stable complexes with copper ions (Cu2+). Semax is a synthetic peptide analogue of Adrenocorticotropic Hormone (ACTH) (4-10). Semax has immunomodulatory, pro intellectual, and neuroprotective activities. Semax can be used for research on central nervous system diseases such as Alzheimer's disease and cerebral ischemia.

Semax is a heptapeptide with the following amino acid sequence: Met-Glu-His-Phe-Pro-Gly-Pro (MEHFPGP, single letter form).

Research of Semax

In the vast universe of neuroscience, there is a small asteroid named Semax emitting a dazzling light. As an artificially synthesized peptide based on the improvement of endorphin structure, Semax, although born in a laboratory in Russia, quickly attracted the attention of the global scientific research community. Its potential in brain protection, cognitive enhancement, and psychological adaptation has made it a hot topic in the field of nerve regeneration and recovery. Semax increases levels of brain-derived neurotrophic factor (BDNF). Semax also helps reduce the breakdown of enkephalins.

Omizzur synthesis strategy for Semax: Typically, Fmoc or Boc solid-phase synthesis is used, with resin as the carrier, sequentially coupling protected amino acids, and finally cutting and deprotection.

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Semax Preclinical/Clinical Research Hotspots:

Neurodegenerative diseases: demonstrated to reduce A β deposition and protect dopaminergic neurons in Alzheimer's and Parkinson's disease models.

Cognitive enhancement: Studies on healthy individuals have shown that it can improve working memory, attention, and executive function without addiction.

Changxinguan intervention: preliminary research suggests that it has the potential to improve "brain fog", chronic fatigue and cognitive decline.

Safety: Due to structural optimization, hormone like activity has been removed (without HPA axis activation), and no significant glucocorticoid side effects have been observed; The main risks stem from impurities (such as endotoxins) or oxidative deactivation caused by improper storage

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